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Amino Acid Therapy Migraine

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Amino Acid Therapy Migraine

Amino acid therapy migraine provides a natural and safe method to alleviate acute headache pain. Additionally, it’s an excellent preventive measure as it optimizes neurotransmitter function in the brain and central nervous system. Amino acids such as 5-HTP, GABA and L-tryptophan are essential in reducing inflammation which has been linked to migraine attacks.

Migraine is a common, disabling disorder characterized by intense headaches that usually strike one side of the head. Other symptoms may include nausea, vomiting, light sensitivity, tinnitus and photophobia. Migraine occurs more frequently among women between 25-45 years of age, and it can be highly debilitating for both sufferers and their family members.

The biochemistry of migraine is intricate, involving several neurotransmitter systems that interact in synaptic pathways. Serotonin plays a major role in migraine, but other neurotransmitters like adenosine triphosphate (ATP), tyrosine, glutamate and dopamine may also be involved.

Migraine pain is typically caused by an abnormal release of neurotransmitters in the brain. This can occur due to various factors such as toxins, changes to one’s endocrine system or problems within vascular networks.

5-Hydroxytryptophan has been found to decrease plasma 5-hydroxyindoleacetic acid, a serotonin metabolite, in migraine sufferers. This has been observed in several studies and it may be beneficial to administer 5-HTP daily as an antimicrobial medication to decrease migraine incidence.

Unfortunately, research has demonstrated that the amount of 5-HTP needed to sustain a normal level of serotonin in the brain is too low for this approach to be effective as an abortive treatment. Furthermore, it’s difficult to accurately determine how much 5-HTP gets metabolized into serotonin throughout various organs such as the gut, liver or systemic circulation; thus oral administration of 5-HTP typically fails to reduce migraine pain symptoms.

Kynurenine pathway metabolites have been implicated in the neurophysiology of migraine, as they suppress oxidative stress and are believed to be involved in glutamate signaling within the CNS. Altered levels of kynurenine have been observed both chronic and acute migraine patients, suggesting it could serve as a therapeutic target for this disorder.

Animal models of migraine have demonstrated that tyramine downregulates both iGluRs and mGluRs, leading to reduced nitroglycerin-evoked hyperalgesia and cortical spreading depression.

Tyramine is an amino acid naturally present in many foods that has the ability to vaso-activate. It plays an essential role in the conversion of tyrosine into epinephrine, the active hormone produced by the inner portion of the adrenal glands. This process has been known to initiate a chain reaction which causes selective cerebral vasoconstriction followed by rebound dilation of cranial vessels and the familiar headache pain associated with migraine.

Kynurenine pathway metabolites such as kynurenine amides and acetyl-kynurenine have been studied as a potential therapeutic approach for migraine. These compounds may act on iGluRs, mGluRs and nicotinic receptors which are believed to be involved in activating glutamatergic synapses within the brain.

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- Welcome, SoundTherapy.com lowers anxiety 86%, pain 77%, and boosts memory 11-29%. Click on the brain to sign up or share with buttons below to help others:
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